Transcriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Mice

Persistent Link:
http://hdl.handle.net/10150/612996
Title:
Transcriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Mice
Author:
Larmonier, Claire B.; Shehab, Kareem W.; Laubitz, Daniel; Jamwal, Deepa R.; Ghishan, Fayez K.; Kiela, Pawel R.
Affiliation:
Univ Arizona, Hlth Sci Ctr, Steele Childrens Res Ctr, Dept Pediat; Univ Arizona, Hlth Sci Ctr, Dept Immunobiol
Issue Date:
2016-04-22
Publisher:
AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC
Citation:
Transcriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Mice 2016, 291 (17):8918 Journal of Biological Chemistry
Journal:
Journal of Biological Chemistry
Rights:
© 2016 by The American Society for Biochemistry and Molecular Biology, Inc.
Collection Information:
This item from the UA Faculty Publications collection is made available by the University of Arizona with support from the University of Arizona Libraries. If you have questions, please contact us at repository@u.library.arizona.edu.
Note:
12 month Embargo
ISSN:
0021-9258; 1083-351X
DOI:
10.1074/jbc.M116.714386
Keywords:
ADP-RIBOSE POLYMERASE; REGULATORY T-CELLS; NF-KAPPA-B; EXPERIMENTAL COLITIS; FECAL MICROBIOTA; PARP-1; BINDING; INFLAMMATION; INHIBITION; APOPTOSIS
Version:
Final published version
Sponsors:
This work was supported by National Institutes of Health Grants R37DK033209 (to F. K. G.) and 1K01DK099268 (to C. B. L.) through the NIDDK. The authors declare that they have no conflicts of interest with the contents of this article. The content is solely the responsibility of the authors and does not necessarily represent the official views of the National Institutes of Health.
Additional Links:
http://www.jbc.org/lookup/doi/10.1074/jbc.M116.714386

Full metadata record

DC FieldValue Language
dc.contributor.authorLarmonier, Claire B.en
dc.contributor.authorShehab, Kareem W.en
dc.contributor.authorLaubitz, Danielen
dc.contributor.authorJamwal, Deepa R.en
dc.contributor.authorGhishan, Fayez K.en
dc.contributor.authorKiela, Pawel R.en
dc.date.accessioned2016-06-14T01:50:05Z-
dc.date.available2016-06-14T01:50:05Z-
dc.date.issued2016-04-22-
dc.identifier.citationTranscriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Mice 2016, 291 (17):8918 Journal of Biological Chemistryen
dc.identifier.issn0021-9258-
dc.identifier.issn1083-351X-
dc.identifier.doi10.1074/jbc.M116.714386-
dc.identifier.urihttp://hdl.handle.net/10150/612996-
dc.description.sponsorshipThis work was supported by National Institutes of Health Grants R37DK033209 (to F. K. G.) and 1K01DK099268 (to C. B. L.) through the NIDDK. The authors declare that they have no conflicts of interest with the contents of this article. The content is solely the responsibility of the authors and does not necessarily represent the official views of the National Institutes of Health.en
dc.language.isoenen
dc.publisherAMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INCen
dc.relation.urlhttp://www.jbc.org/lookup/doi/10.1074/jbc.M116.714386en
dc.rights© 2016 by The American Society for Biochemistry and Molecular Biology, Inc.en
dc.subjectADP-RIBOSE POLYMERASEen
dc.subjectREGULATORY T-CELLSen
dc.subjectNF-KAPPA-Ben
dc.subjectEXPERIMENTAL COLITISen
dc.subjectFECAL MICROBIOTAen
dc.subjectPARP-1en
dc.subjectBINDINGen
dc.subjectINFLAMMATIONen
dc.subjectINHIBITIONen
dc.subjectAPOPTOSISen
dc.titleTranscriptional Reprogramming and Resistance to Colonic Mucosal Injury in Poly(ADP-ribose) Polymerase 1 (PARP1)-deficient Miceen
dc.typeArticleen
dc.contributor.departmentUniv Arizona, Hlth Sci Ctr, Steele Childrens Res Ctr, Dept Pediaten
dc.contributor.departmentUniv Arizona, Hlth Sci Ctr, Dept Immunobiolen
dc.identifier.journalJournal of Biological Chemistryen
dc.description.note12 month Embargoen
dc.description.collectioninformationThis item from the UA Faculty Publications collection is made available by the University of Arizona with support from the University of Arizona Libraries. If you have questions, please contact us at repository@u.library.arizona.edu.en
dc.eprint.versionFinal published versionen
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